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Category: Computer softwareAttenuation of diltiazem-induced abnormal calcium transients in cultured neonatal rat cardiomyocytes by amiodarone.
Amiodarone therapy is limited by its side effects, including cardiac arrhythmias, which may be mediated by the drug’s effects on the cellular electrophysiology of the heart. Amiodarone blocks L-type calcium channels and increases transient outward K(+) currents of the cardiac cell membrane. The results of our study, however, showed that in amiodarone-treated cardiomyocytes, amiodarone significantly inhibited the transient outward K(+) current and attenuated the intracellular Ca(2+) concentration increase during the action potential plateau. In addition, amiodarone slightly suppressed the L-type calcium channel current, which might be due to the multidrug binding and lead to the abnormal voltage-dependent Ca(2+) currents in cardiomyocytes. These results indicate that amiodarone inhibits transient outward K(+) currents and transient intracellular Ca(2+) concentration increase during the action potential plateau by a mechanism involving multiple drug effects on the K(+) channel and the Ca(2+) channel.increments(‘id’);
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